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LNCRNA Illumina sequencing results associated with renal fibrosis disease progression

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Jun 06, 2024 version files 91.05 GB

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Abstract

Objective: To screen and identify long noncoding RNAs (lncRNAs) involved in the progression of renal fibrosis and to explore their functions. Methods: We used unilateral ureteral obstruction (UUO) to establish a rat model of renal fibrosis. The animals were randomly divided into four groups: control, model group at 2 weeks (MOD-2), MOD-4, and MOD-6. Renal function was monitored by measuring the levels of conventional biochemical biomarkers. Differentially expressed lncRNAs (DE-lncRNAs) between the control and model groups were screened, and their functions were annotated using Gene Ontology and Kyoto Encyclopedia of Genes and Genomes analysis. Gene interaction networks were visualized using Cytoscape.

Results: Transcriptomic analysis revealed 243 DE-lncRNAs (82 upregulated, 161 downregulated) in the MOD-2 group compared with the control group, 216 DE-lncRNAs (95 upregulated, 121 downregulated) in the MOD-4 group, and 70 DE-lncRNAs (39 upregulated, 31 downregulated) in the MOD-6 group. Compared with the control group, the model groups were enriched in target genes involved in extracellular matrix–receptor interaction, arginine and proline metabolism, arachidonic acid metabolism, glutathione metabolism, peroxisome proliferator-activated receptor signaling, Janus kinase–signal transducer and activator of transcription signaling, phosphoinositide-3-kinase–Akt signaling, and calcium signaling. 

Conclusion: We speculate that the six upregulated DE-lncRNAs in the model groups may be involved in the occurrence and development of renal fibrosis, while the seven downregulated DE-lncRNAs may have inhibitory effects